Cancer treatments can work in lots of different ways, aiming to kill tumour cells or keep them under control. Ideally they cause tumours to shrink, but they can also be considered successful if they stop tumours growing. But unfortunately, the effects don’t always last forever.
Scientists have developed an entirely new way to look at tumours. And it’s helped them solve the mystery of how some pancreatic tumours develop.
Anh Hoang Le, a PhD student at the Cancer Research UK Beatson Institute in Glasgow, studies two proteins that we know curiously little about: CYRI-A and CYRI-B.
We look at the research made possible by people who donate their bodies after their death, including helping scientists understand how advanced cancers spread.
It’s hard to talking about cancers ‘knowing’ something, but they can have predictable patterns of spread. And scientists are beginning to understand why.
Our international team of scientists are tackling a fundamental question about how cancer develops: why do some gene faults only cause cancer in certain organs?
Inflammation can prevent infections and helps repair injuries. But for people with long-term inflammatory conditions, it can sometimes lead to cancer.
Scientists are taking a leaf out of Darwin’s evolutionary handbook to understand how lung cancer evolves.
Our scientists have uncovered a new marker that’s found on treatment-resistant prostate cancer cells.